Neurobiology Of Disease

Gene Expression Profiles in a Transgenic Animal Model of Fragile X Syndrome

Neurobiology Of Disease / Neurobiology / Molecular Mechanics / Mental Retardation / Brain / Mice / Female / Animals / Male / Animal Model / Fragile X Syndrome / Amyloid Beta / Spatial Distribution / Clinical Sciences / Knockout Mice / Neurosciences / Microarray Data / Gene expression profiling / Amyloid Beta Precursor Protein / Mice / Female / Animals / Male / Animal Model / Fragile X Syndrome / Amyloid Beta / Spatial Distribution / Clinical Sciences / Knockout Mice / Neurosciences / Microarray Data / Gene expression profiling / Amyloid Beta Precursor Protein

Increase in endoplasmic reticulum-associated tissue transglutaminase and enzymatic activation in a cellular model of Parkinson\'s disease

Neurobiology Of Disease / Drug interactions / Enzyme Inhibitors / Transmission Electron Microscopy / Neurobiology / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases / Cell Differentiation / Humans / Endoplasmic Reticulum / Tunicamycin / ER stress / Neurons / Human Brain / Enzyme / Clinical Sciences / Neuroblastoma / Cellular Automaton Model / Degeneration / Retinoic Acid / Tretinoin / Membrane Protein / Enzymatic Activity / Cytoplasm / Neurosciences / Transglutaminases

Differential suppression of seizures via Y2 and Y5 neuropeptide Y receptors

Neurobiology Of Disease / Epilepsy / Hippocampus / Magnesium / Mice / Female / Animals / Male / Temporal Lobe Epilepsy / Animal Model / Synaptic Transmission / Neurons / Clinical Sciences / Neuropeptide Y / Knockout Mice / Neurosciences / Gene Expression Regulation / Female / Animals / Male / Temporal Lobe Epilepsy / Animal Model / Synaptic Transmission / Neurons / Clinical Sciences / Neuropeptide Y / Knockout Mice / Neurosciences / Gene Expression Regulation

Aquaporin-4 deletion in mice reduces encephalopathy and brain edema in experimental acute liver failure

Neuroscience / Neurobiology Of Disease / Neurobiology / Aquaporins (AQP) / Mice / Animals / Astrocytes / Clinical Sciences / Hepatic Encephalopathy / Analysis of Variance / Time Factors / Aquaporin / Acetaminophen / model of Acute liver failure / Thioacetamide / Neurosciences / Brain Edema / Gene Expression Regulation / Brain Diseases / Animals / Astrocytes / Clinical Sciences / Hepatic Encephalopathy / Analysis of Variance / Time Factors / Aquaporin / Acetaminophen / model of Acute liver failure / Thioacetamide / Neurosciences / Brain Edema / Gene Expression Regulation / Brain Diseases

SEMA4D compromises blood-brain barrier, activates microglia, and inhibits remyelination in neurodegenerative disease

Neurobiology Of Disease / Clinical Sciences / Neurosciences

Disrupted cross-laminar cortical processing in β amyloid pathology precedes cell death

Neurobiology Of Disease / Fear / Fourier Analysis / Humans / Mutation / Cerebral Cortex / Mice / Animals / Cell Death / Neurons / Clinical Sciences / Thallium / Presenilin-2 / Age Factors / Alzheimer Disease / Neurosciences / Amyloid Beta Precursor Protein / Cerebral Cortex / Mice / Animals / Cell Death / Neurons / Clinical Sciences / Thallium / Presenilin-2 / Age Factors / Alzheimer Disease / Neurosciences / Amyloid Beta Precursor Protein

Molecular networks perturbed in a developmental animal model of brain injury

Neurobiology Of Disease / Cell Cycle / Transcription Regulation / DNA damage / Neurobiology / Gene expression / Cerebral Cortex / Mice / Animals / Animal Model / Brain injury / Clinical Sciences / Protein Metabolism / Neurodegenerative Disease / Nervous System / Neurosciences / Brain injuries / Gene expression / Cerebral Cortex / Mice / Animals / Animal Model / Brain injury / Clinical Sciences / Protein Metabolism / Neurodegenerative Disease / Nervous System / Neurosciences / Brain injuries

Partial mitochondrial complex I inhibition induces oxidative damage and perturbs glutamate transport in primary retinal cultures

Neurobiology Of Disease / Free Radicals / Oxidative Stress / Mitochondria / Neurobiology / Energy Metabolism / Free Radical / Glutamate / Mitochondrial DNA / Animals / Cell Death / Retina / Glial Cell / Neurons / Lipid peroxidation / Clinical Sciences / Rats / Oxidative Damage / Degeneration / Retinal Ganglion Cells / Wistar Rats / Neurosciences / Glutamic Acid / Glutamate Transporter / Complex I / Energy Metabolism / Free Radical / Glutamate / Mitochondrial DNA / Animals / Cell Death / Retina / Glial Cell / Neurons / Lipid peroxidation / Clinical Sciences / Rats / Oxidative Damage / Degeneration / Retinal Ganglion Cells / Wistar Rats / Neurosciences / Glutamic Acid / Glutamate Transporter / Complex I

SEMA4D compromises blood-brain barrier, activates microglia, and inhibits remyelination in neurodegenerative disease

Neurobiology Of Disease / Mice / Blood brain barrier / Female / Animals / Male / Monoclonal Antibodies / Clinical Sciences / Rats / Experimental Autoimmune Encephalomyelitis / Neurosciences / Semaphorins / Male / Monoclonal Antibodies / Clinical Sciences / Rats / Experimental Autoimmune Encephalomyelitis / Neurosciences / Semaphorins

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