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Caspase
Caspase
Interaction with substrate sensitises caspase-3 to inactivation by hydrogen peroxide
Catalysis / Apoptosis / Caspases / Humans / Enzyme / Caspase / Hydrogen Peroxide / Second Order / Active site / Substrate Specificity / Oxidation / Cysteine / Oxidation-Reduction / Catalytic Activity / Biochemistry and cell biology / Caspase / Hydrogen Peroxide / Second Order / Active site / Substrate Specificity / Oxidation / Cysteine / Oxidation-Reduction / Catalytic Activity / Biochemistry and cell biology
Interaction with substrate sensitises caspase-3 to inactivation by hydrogen peroxide
Catalysis / Apoptosis / Caspases / Humans / Enzyme / Caspase / Hydrogen Peroxide / Second Order / Active site / Substrate Specificity / Oxidation / Cysteine / Oxidation-Reduction / Catalytic Activity / Biochemistry and cell biology / Caspase / Hydrogen Peroxide / Second Order / Active site / Substrate Specificity / Oxidation / Cysteine / Oxidation-Reduction / Catalytic Activity / Biochemistry and cell biology
PIGA (N,N-Di-n-butyl-5-chloro-2-(4-chlorophenyl)indol-3-ylglyoxylamide), a New Mitochondrial Benzodiazepine-Receptor Ligand, Induces Apoptosis in C6 glioma Cells
Mitochondria / DNA damage / Apoptosis / Caspases / Glioma / Animals / Caspase / Rats / Time Factors / Receptors / Antineoplastic Agents / Ligands / Biochemistry and cell biology / Animals / Caspase / Rats / Time Factors / Receptors / Antineoplastic Agents / Ligands / Biochemistry and cell biology
Cisplatin compromises myocardial contractile function and mitochondrial ultrastructure: Role of endoplasmic reticulum stress
Physiology / Endoplasmic Reticulum Stress / Apoptosis / Hemodynamics / Mice / Animals / Male / Endoplasmic Reticulum / Heart / Medical Physiology / Caspase / Cisplatin / Myocardium / Cell Shape / Physiological Stress Markers / Antineoplastic Agents / Animals / Male / Endoplasmic Reticulum / Heart / Medical Physiology / Caspase / Cisplatin / Myocardium / Cell Shape / Physiological Stress Markers / Antineoplastic Agents
Multiple, Parallel Cellular Suicide Mechanisms Participate In Photoreceptor Cell Death
Complement activation / Metabolism / Oxidative Stress / Autophagy / Apoptosis / Gene expression / Glucose Metabolism / Laser Capture Microdissection / Mice / Animals / Cell Death / Retinal Degeneration / Animal Model / Caspase / Optometry and Ophthalmology / Time Factors / Degeneration / Environmental Stress / Complement / Temporal Analysis / Genetic Markers / Neurosciences / Time Course / Gene Expression Regulation / Gene expression / Glucose Metabolism / Laser Capture Microdissection / Mice / Animals / Cell Death / Retinal Degeneration / Animal Model / Caspase / Optometry and Ophthalmology / Time Factors / Degeneration / Environmental Stress / Complement / Temporal Analysis / Genetic Markers / Neurosciences / Time Course / Gene Expression Regulation
Vascular Leak in a Rat Model of Preeclampsia
STEROIDS / Pregnancy / Humans / Female / Animals / Male / American / Clinical Sciences / Caspase / Rats / Light Intensity / Time Dependent / Rat Model / Vascular Permeability / Pre Eclampsia / Male / American / Clinical Sciences / Caspase / Rats / Light Intensity / Time Dependent / Rat Model / Vascular Permeability / Pre Eclampsia
Multiple, Parallel Cellular Suicide Mechanisms Participate In Photoreceptor Cell Death
Complement activation / Metabolism / Oxidative Stress / Autophagy / Apoptosis / Gene expression / Glucose Metabolism / Laser Capture Microdissection / Mice / Animals / Cell Death / Retinal Degeneration / Animal Model / Caspase / Optometry and Ophthalmology / Time Factors / Degeneration / Environmental Stress / Complement / Temporal Analysis / Genetic Markers / Neurosciences / Time Course / Gene Expression Regulation / Gene expression / Glucose Metabolism / Laser Capture Microdissection / Mice / Animals / Cell Death / Retinal Degeneration / Animal Model / Caspase / Optometry and Ophthalmology / Time Factors / Degeneration / Environmental Stress / Complement / Temporal Analysis / Genetic Markers / Neurosciences / Time Course / Gene Expression Regulation
Arachidonic acid induces brain endothelial cell apoptosis via p38-MAPK and intracellular calcium signaling
Inflammation / Calcium / Mitochondria / Apoptosis / Gene Silencing / RNA interference / Microvascular Physiology / Brain / Humans / Endothelial Cells / Lipoxygenases / Calcium Signaling / Mitogen Activated Protein Kinase / Clinical Sciences / Caspase / Cell Proliferation / Arachidonic Acid / RNA interference / Microvascular Physiology / Brain / Humans / Endothelial Cells / Lipoxygenases / Calcium Signaling / Mitogen Activated Protein Kinase / Clinical Sciences / Caspase / Cell Proliferation / Arachidonic Acid
Arachidonic acid induces brain endothelial cell apoptosis via p38-MAPK and intracellular calcium signaling
Inflammation / Calcium / Mitochondria / Apoptosis / Gene Silencing / RNA interference / Microvascular Physiology / Brain / Humans / Endothelial Cells / Lipoxygenases / Calcium Signaling / Mitogen Activated Protein Kinase / Clinical Sciences / Caspase / Cell Proliferation / Arachidonic Acid / RNA interference / Microvascular Physiology / Brain / Humans / Endothelial Cells / Lipoxygenases / Calcium Signaling / Mitogen Activated Protein Kinase / Clinical Sciences / Caspase / Cell Proliferation / Arachidonic Acid
Levosimendan preserves the contractile responsiveness of hypoxic human myocardium via mitochondrial KATP channel and potential pERK 1/2 activation
Mitochondria / Apoptosis / Humans / Human right / Cytochrome C / Mitogen Activated Protein Kinase / Caspase / European / Hydrazones / Oxygen / Myocardium / Western blot / Muscle contraction / Potassium Channels / Mechanism of action / Isoproterenol / Mitogen Activated Protein Kinase / Caspase / European / Hydrazones / Oxygen / Myocardium / Western blot / Muscle contraction / Potassium Channels / Mechanism of action / Isoproterenol
Arsenic Trioxide Inhibits Neuroblastoma Growth in Vivo and Promotes Apoptotic Cell Death in Vitro
Apoptosis / Cell Division / Caspases / Cell line / Cell Differentiation / Humans / Cell Death / Biochemical / Differentiation / Arsenic trioxide / Caspase / Neuroblastoma / Retinoic Acid / Clinical Study / Tretinoin / Oxides / Thiazoles / Cell Survival / Tumor Growth / Antineoplastic Agents / Arsenicals / Acute Promyelocytic Leukemia / Nude mice / Biochemistry and cell biology / Humans / Cell Death / Biochemical / Differentiation / Arsenic trioxide / Caspase / Neuroblastoma / Retinoic Acid / Clinical Study / Tretinoin / Oxides / Thiazoles / Cell Survival / Tumor Growth / Antineoplastic Agents / Arsenicals / Acute Promyelocytic Leukemia / Nude mice / Biochemistry and cell biology
Levosimendan preserves the contractile responsiveness of hypoxic human myocardium via mitochondrial KATP channel and potential pERK 1/2 activation
Mitochondria / Apoptosis / Humans / Human right / Cytochrome C / Mitogen Activated Protein Kinase / Caspase / European / Hydrazones / Oxygen / Myocardium / Western blot / Muscle contraction / Potassium Channels / Mechanism of action / Isoproterenol / Mitogen Activated Protein Kinase / Caspase / European / Hydrazones / Oxygen / Myocardium / Western blot / Muscle contraction / Potassium Channels / Mechanism of action / Isoproterenol
Ultrasonic low-energy treatment
Leukemia / Oxidative Stress / Mitochondria / Apoptosis / Caspases / Humans / Glutathione / Low Energy Buildngs / Ultrasonic therapy / Membrane Lipids / Caspase / Experimental / Singlet Oxygen / Mechanism of action / Cell Survival / Histidine / Mannitol / DNA fragmentation / Humans / Glutathione / Low Energy Buildngs / Ultrasonic therapy / Membrane Lipids / Caspase / Experimental / Singlet Oxygen / Mechanism of action / Cell Survival / Histidine / Mannitol / DNA fragmentation
Hemigramicidin–TEMPO conjugates: Novel mitochondria-targeted anti-oxidants
Nursing / Free Radicals / Phospholipids / Mitochondria / Apoptosis / Critical Care Medicine / Free Radical / Antioxidants / Humans / Intestinal Mucosa / Reactive Oxygen Species / Chronic Disease / Animals / Biochemical / Lipid peroxidation / Clinical Sciences / Caspase / Anti-Bacterial Agents / Rats / Public health systems and services research / Oxidative Damage / BIOCHEMICAL PHARMACOLOGY / Hemorrhagic Shock / Critical Illness / Gramicidin / Critical Care Medicine / Free Radical / Antioxidants / Humans / Intestinal Mucosa / Reactive Oxygen Species / Chronic Disease / Animals / Biochemical / Lipid peroxidation / Clinical Sciences / Caspase / Anti-Bacterial Agents / Rats / Public health systems and services research / Oxidative Damage / BIOCHEMICAL PHARMACOLOGY / Hemorrhagic Shock / Critical Illness / Gramicidin
NADH fluorescence as a photobiological metric in 5-aminolevlinic acid (ALA)-photodynamic therapy
Fluorescence / Photobiology / Photodynamic Therapy / Signal Transduction / Humans / Cell Death / Caspase / Optical physics / Photochemotherapy / Time Factors / Necrosis / NAD / Cell Survival / Electrical And Electronic Engineering / Aminolevulinic Acid / Cell Death / Caspase / Optical physics / Photochemotherapy / Time Factors / Necrosis / NAD / Cell Survival / Electrical And Electronic Engineering / Aminolevulinic Acid
Antioxidants block proteasome inhibitor function in endometrial carcinoma cells
Vitamins / Apoptosis / Western blotting / Antioxidants / Ubiquitin / Humans / Female / Vitamin E / Caspase / Coumarins / Ascorbic Acid / Boronic Acids / Endometrial carcinoma / Cell Survival / Anti Cancer Drugs / Anti Cancer / Antipyrine / PROTEASOME INHIBITOR / Cysteine Proteinase Inhibitors / Humans / Female / Vitamin E / Caspase / Coumarins / Ascorbic Acid / Boronic Acids / Endometrial carcinoma / Cell Survival / Anti Cancer Drugs / Anti Cancer / Antipyrine / PROTEASOME INHIBITOR / Cysteine Proteinase Inhibitors
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